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Moreover, elevated insulin levels resulting from hyperinsulinemia trigger insulin-like growth factor (IGF) signaling, activating key pathways such as phosphoinositide 3-kinase (PI3K)/protein kinase B (Akt)/mammalian target of rapamycin (mTOR) and mitogen-activated protein kinase (MAPK) [26]

*Data are mean (SD) number of sunburn cells

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Statements Author contributions NA and HA conceptualized the idea of this research

IGF-1, Insulin-like growth factor-1

Scalise et al., 2018b)