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Genetic Predispositions in GLP-1 and GIP Pathways: Why Individual Response Varies Genetic variations in GLP-1R (glucagon-like peptide-1 receptor) and GIPR genes influence how efficiently individuals' cells respond to these medications

Selenium can boost the anti-ferroptosis activity of GPX4 via selenocysteine residues at position-46 [53], and exogenous selenium supplementation inhibited ferroptosis in a mouse model of intracerebral haemorrhage [53,54,55]

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