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The observed failure of drugs targeting S-glutathionylation in clinical trials regarding ovarian cancer might be explained by resistance mechanisms such as increased/decreased rates of drug efflux/influx as cellular redox status affects transporters function or resistance to apoptosis through caspase-3 S-glutathionylation [73]

In an announcement of the study on the Vanderbilt website, lead author Khrysta Baig described this as a purely hypothetical scenario, but one that underscores that at current prices, these medications cannot be the only way or even the main way we address obesity as a society. In a more conservative estimate, Bhaig and coauthors consider a case where only about 10% of those eligible for obesity treatment opted for semaglutide, which would result in $27 billion in new costs

Targeting the Melanocortin System to Treat Hereditary Obesity Table 3: Selected genetic causes of obesity Setmelanotide, an MC4R agonist and first-in-class treatment for hereditary obesity Drug Development Pipeline for Genetic Causes of Obesity 6

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Secondly, prodrug strategies can efficiently deliver drugs to tumors with reduced side effects

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